1670 ROUTE 34 N. 3R FLOOR SUITE 3C WALL • NJ 07727
1670 ROUTE 34 N. 3R FLOOR SUITE 3C WALL • NJ 07727
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Emanuel Revici

The Work of Emanuel Revici MD

  • Born in Romania in 1896, deceased in 1997 
  • A brilliant scientist and medical doctor
  • Spent decades doing research in physiopathology
  • Published his seminal book in 1961 describing a previously unknown homeostatic control mechanism
  • Revici’s license to practice medicine was revoked in 1993  
  • Revici’s contributions have been marginalized in conventional medicine despite profound implications of his work 

Anabolic – Catabolic Imbalances 

  • A common, but rarely recognized cause of chronic illness
  • Also referred to as Anaerobic/Dysaerobic imbalance
  • The Anaerobic imbalance is also known as the “Warburg Effect”- or anerobic glycolysis – also associated with “reductive stress”
  • The Dysaerobic imbalance is associated with “oxidative stress”

Balance Between Anabolic and Catabolic Tendencies is Vital for Health

  • In a balanced state there are sufficient sterols and fatty acids to allow the cells to produce energy in an efficient manner 
  • Enough sterols to control oxidative stress 
  • Enough fatty acids to facilitate oxidative metabolism 
  • Appropriate membrane permeability 
  • Appropriate pH balance between intracellular and extracellular compartments 

Anabolic/Catabolic Balance is Achieved Toward the Middle of the Curve

A Balance Between Fatty Acids and Sterols

Anabolic (Anaerobic)

  • Caused by excessive sterol activity within lipid bilayer of cell and mitochondrial membranes 
  • The relative lack of free fatty acids available to bond with oxygen leads to an anaerobic metabolic state 
  • This anaerobic state leads to increased growth of connective tissue and dedifferentiated growth
  • Decreased membrane permeability 
  • Excess tissue acidity and excess systemic alkalinity

Too Many Sterols

Anaerobic Patterns

  • Common Symptoms -constipation, sleepy fatigue, polyuria, depression, burning pain (tissue acidity), adverse response to prednisone 
  • Lab Findings – may have -low CRP, low serum cholesterol, high estrogen, high progesterone, high cortisol, increased symptoms during mid-luteal phase of cycle
  • Metabolic Tests – low urine SG < 15, high urine pH > 6.2, low saliva pH < 6.7, slow absorption of skin wheel 
  • Benefit from relatively low cholesterol foods
  • Benefit from sulfur products

Anaerobic Overview

  • Anaerobic (lack of oxygen- also known as anaerobic glycoysis = Warburg Effect) – Lactic acid is a product of anaerobic metabolism 
  • Treatment – Relatively low cholesterol diet, Oxytonic (sodium thiosulfate), Oxygenic A ,Taurine.

Catabolic (Dysaerobic)

  • Caused by excessive fatty acid activity within lipid bilayer of cell and mitochondrial membranes 
  • The relative lack of sterols available to bind to fatty acids exposes these fatty acids to excessive oxidative stress 
  • This dysaerobic state leads to decreased growth of connective tissue and degeneration
  • Increased membrane permeability 
  • Excess tissue alkalinity and excess systemic acidity

Too Many Fatty Acids, Not Enough Sterols

Dysaerobic Patterns

  • Common Symptoms – diarrhea, burnt out fatigue, insomnia, anxiety oliguria, sharp pain (tissue alkalinity) responds well to prednisone
  • Lab Findings – may have -high CRP, high serum cholesterol, low estrogen, low progesterone, low cortisol, increased symptoms during menses
  • Metabolic Tests – high urine SG > 15, low urine pH < 6.1, high saliva pH > 6.7, rapid absorption of skin wheel 
  • Benefit from relatively high cholesterol foods
  • Reacts poorly to sulfur products – dysaerobic symptoms

Dysaerobic Overview

  • Dysaerobic (dysfunctional use of oxygen) = excess FA Deficient in sterols = chloride pulled into FA leaving behind carbonates that cause tissue alkalinity- Saliva alkaline  Treatment – High Cholesterol Diet, Oxygenic D+(glycerol and butanol), Oxygenic D, Glycine

Therapeutic Agents Effects on Imbalances

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